Student #1
During my nurse residency program, I participated in a project that collects objectivee data on how effective our new Intravenous line(IV) assessment guideline was in preventing IV-associated infections. We collected data through pre and post-surveys that were sent out to the nurses. The survey questions were consisted of binary and discrete data(eg. yes/no, rating from a scale of 0 to 10, infection counts, etc). And we gave about a month between the pre and post-survey. It was not the most rigorous experiment but our team managed to get enough responses back to compare the results. We turned the results into bar charts so it was easier to visualize any changes. We concluded that there were decreased cases of IV-associated complications after the new guideline implementation.
Student #2
One situation in which I was responsible for collecting data was during my internship in which I held the role of Patient Experience Intern. The majority of my responsibilities included surveying current and prior patients of the hospital’s emergency department to obtain satisfaction levels regarding their experience while staying at the hospital, with the data collected to be used to adjust the department’s standard operating procedure. All data collected was done via surveys distributed to patients through the patient portal or by phone call. Specific data collected included qualitative variables such as age, gender, race, ethnicity, and yes/no questions pertaining to satisfaction level, for example, “were you satisfied with the overall level of care you received?”. Other data obtained was quantitative, including age, length of stay, length of wait time to triage, length of wait time to be admitted, and length of time waited to be assessed/treated by a physician or other provider. As I was only responsible for collecting data and my internship with the hospital came to an end prior to the completion of data collection, I cannot comment on the mean, median, or mode of the data nor the center and spread of the data.
The topic for this discussion
Student Initial post
The article I chose was written about a study which looked into the long-term cognitive effects individuals experience after being infected with COVID-19. The study used mice and “explored how mild respiratory infections of SARS-CoV-2 could lead to neuroinflammation and subsequent brain damage through multilineage neural cell dysregulation”. Investigators infected the study mice with COVID and observed the effect on different parts of the body, specifically the brain. What they found was “no SARS-CoV-2 in the brain but found signs of neuroinflammation in elevated levels of chemokines in cerebrospinal fluid and serum, each with a distinct time course”. They then looked into whether this finding of neuroinflammation & its related brain fog could lead to the development of a cure. “Findings from the study by Fernández-Castañeda and colleagues support the testing of microglial modulators to treat Covid-related brain fog” as well as suggest that looking into upstream regulators of microglial activation could be beneficial. In addition, the study deemed CCL11 as a biomarker which, if verified by another study, can be used to identify patients with covid-related cognitive impairment by looking at levels in plasma or cerebrospinal fluid.
I chose this study because I have heard many people within my life talk about feeling as though their head is foggy for weeks after having COVID-19 and figured it is interesting to look into long-term effects on the brain after being infected. I had always believed that there was a correlation between the two, and this study & its findings help to prove it.
Any individuals who had been previously infected with COVID-19 as well as anybody at risk of developing it (aka anyone living in a country impacted by COVID) would be interested in this study & its findings. Knowing the fact that the infection can cause long-term neurological issues is important as people may take extra preventative measures.
I believe the findings of this study, as many previously infected individuals have long-term effects and feel that they haven’t fully recovered. It makes sense that there is a deeper impact, involving the brain & its functioning ability.
Based on the results, I would be interested to hear if the newer variants have as much of a long-term neurological affect as the first variant that had been researched. I would be willing to bet that the more serious variants have similar effects, whereas the less severe variants may not.
https://www.nejm.org/doi/full/10.1056/NEJMcibr2210069?query=featured_coronavirus
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